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AKC REGISTRATION FRAUD

 

KESANG CAMP PUPPIES AND ADULTS FOR ADOPTION

 

TIBETAN MASTIFF HYPOALLERGENIC THERAPY DOGS

 

TIBETAN MASTIFFS SEARCH AND RESCUE DOGS

 

TIBETAN MASTIFFS FOR OBEDIENCE | SPECIALTY

TRAINING

 

TIBETAN MASTIFFTSANG-KHYI VERSUSĀ DO-KHYI

 

TIBETAN MASTIFF GENETIC HEALTH | CIDN REPORTS

 

THE TIBETAN MASTIFF AS LIVESTOCK GUARDIANS

 

TIBETAN MASTIFF AS MILITARY WORKING DOGS

 

KESANG CAMP CONTRACT

KESANG CAMP TIBETAN MASTIFFS

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There are only a few health problems associated with the Tibetan Mastiff. Otitus, Hypothyroidism, Entropia and Hip dysplasia include these relatively rare health problems. Rarest of all, has been a few unknown seizure reports within the breed. Canine Inherited Demyelinative Neuropathy (CIDN) was a nerve disorder that came from one particular line in the early 1980's. Fifteen dogs from the Ausable line were test bred carriers of CIDN which always developed before 12 weeks of age and was most evident between 7 to 10 weeks of age. CIDN affected the nerve fibers that run between the spinal cord and the muscles, causing a degeneration of the protective nerve covering (myelin). This loss of insulation caused impulses from the brain to be lost and caused failure in the hind limbs or forelimbs and was similar to muscular dystrophy in humans. At this time, there are no affected CIDN puppies being reported throughout the world. This fact has been verified by studies done at Cornell University, who ended their study on CIDN in the early 1990's without ever having identified the actual gene defect itself as admitted by Dr. Barry Cooper in 1999 because there were no longer any living carriers in which to test breed. CIDN has long since disappeared from this breed. Since the 1987 test breeding of Ausables Khan to Ausables Koko of Talimer, their have been no verified instances of CIDN cropping back up within this breed. Since the 1980's, all CIDN test breeders have admitted that they have been unable to reproduce CIDN affected puppies and have since retired. Therefore, I don't believe that CIDN was ever a breed specific genetic disease, but rather something else that was related strictly to Ausables line-breeding program. I also believe that Cornell University's genetics department was hasty and irresponsible when declaring CIDN to be a TM breed specific genetic disease without ever having identified the actual gene defect itself in light of the fact that this alleged disease has vanished along with the fifteen dogs who were all from one and the same breeder's bloodline which makes me wonder about other causative environmental factors!

Caution: Don't believe anyone's hearsay without supporting medical verification. After 28 years in this breed, neither I, nor any other TM breeder that I know of (aside from Nash or Allen in conjunction with Dr. Cooper) has ever produced or seen a CIDN affected TM. Unfortunately, there are some income motivated breeders out there, primarily of the European persuasion, who are seeking to gain the upper hand by accusing other U.S. breeders of owning CIDN carriers and thereby producing CIDN affected puppies based solely upon the fact that they have had some Ausable lineage within their lines that were not test bred because of dead or nonexistent alleged carriers of CIDN. These unethical persons are merely alleging that retired test breeders have produced untraceable carriers who are still producing affected puppies. Neither of which can be found or verified. Therefore, fraud is the obvious conclusion!


IN PURSUIT OF THE AUTHENTIC TSANG-KHYI